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Intracellular glutathione also followed the trend of GCLC, exhibiting an NFT-dependent increase that was reversed by anti Nrf2-siRNA to levels comparable to those observed in untreated HepG2 cells (Fig

48, 72 We further found that in colonic epithelial cells subjected to colitis causing chemical (DSS treatment), claudin-2 expression is sharply downregulated and that constitutive claudin-2 expression in these cells protected them from DSS-induced cell death

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Later, as GHK accumulates bound copper 2+, GHK-Cu stimulates stem cell differentiation